研究
学会活動
第83回 日本循環器学会学術集会(2019年3月, 神奈川)
- 大石 充.
- ランチョンセミナー40.
健康長寿を叶えるために. - 樋口公嗣.
- Morning Lecture9. 循環器内科医が知っておくべき全身疾患 (リソソーム病).
Fabry Cardiomyopathy. - 樋口公嗣.
- ランチョンセミナー27. 見逃していませんか?循環器疾患におけるファブリー病とムコ多糖類.
心症状から診断できるファブリー病. - Akasaki Y, Ikeda Y, Uchikado Y, Sasaki Y, Higuchi K, Ohishi M.
- Metabolic stress causes sarcopenia via angiotensin II type I receptor signal-induced mitochondrial fission.
- 神田大輔, 池田義之, 佐々木雄一, 薗田剛嗣, 小瀬戸一平, 吉野聡史, 内匠拓朗, 大石 充.
- Malnutrition and non-use clopidogrel additively aggravate prognosis of critical limb ischemia patients who underwent revascularization.
- 徳重明央, 植田真一郎, 大石 充.
- The cardiovascular outcome of cancer survivor in patients with diabetes mellitus with coronary artery disease: from CHD cohort.
- Sasaki Y, Ikeda Y, Akasaki Y, Uchikado Y, Higuchi K, Ohishi M.
- Estrogen/Sirt1/LKB1/AMPK axis plays a crucial role in mediating Rab9-dependent mitophagy and retarding arterial senescence.
- Ojima S, Kubozono T, Kawasoe S, Ohtsubo H, Miyata M, Ohishi M.
- The patients with rheumatoid arthritis had a risk of progression on aortic valve stenosis.
- Ojima S, Kubozono T, Kawasoe S, Miyata M, Miyahara H, Maenohara S, Ohishi M.
- C-reactive protein was significantly associated with arterial stiffness, but not with carotid atherosclerosis in general population.
- 下野洋和, 加治屋 崇, 髙岡順一郎, 宮村明宏, 井上尊文, 二宮登志子, 有村俊博, 厚地良彦, 厚地伸彦, 大石 充.
- Characteristics of recurrent in-stent restenosis (ISR) after second and third generation drug-eluting stent (DES) implantation.
- Yoshimoto I, Oketani N, Iriki Y, Ichiki H, Okui H, Maenosono R, Namino F, Miyata M, Ohishi M.
- Incidence of asymptomatic stroke or hemopericardium in catheter ablation for atrial fibrillation with uninterrupted use of Edoxaban, Rivaroxaban, or Warfarin.
- Imamura S, Miyata M, Kamata R, Nomoto Y, Yoshimoto I, Ohmure K, Chaen H, Ogawa M, Hamasaki S, Yoshihara H, Ohishi M.
- Prognostic predictors in patients with cardiopulmonary arrest: a novel formula to predict death.
- Uchikado Y, Ikeda Y, Sasaki Y, Akasaki Y, Higuchi K, Ohishi M.
- Metabolic stress and angiotensin II causes vascular senescence via AT1R signal-mediate mitochondrial fission.